NAME

Question types


Start with


Question limit

of 48 available terms

Print test

48 Multiple choice questions

  1. hair cell and other cochlera structures dmaged or destroyed
    -dependent on intensity, spectrum of noise, duration of exposure & individual resistance (not necessarily related to loudness of noise)
  2. -acoustic neuroma/vestibular schawnnoma
    -multiple sclerosis
    -cerebovascular accident/stroke
  3. hearing loss and difficutly w/ speech recognition in noisy conditions
  4. -Low-sodium diets
    -Diuretics
    -Sedatives, tranquilizers
    -Vestibular suppressants (e.g., Antivert); drugs that suppress the vestiulus system
    -Endolymphatic shunt surgery
    -Destruction of the labyrinth/cutting the auditory nerve
  5. goiter and profound deafness
  6. base of cochlea (high frequencies)
  7. low frequencies
  8. bile deposits in CNS at cochlear nuclei
  9. transmitted to child in eurtero
    -may manifest itself up to 60 years later, if syphilis shows < 10 yrs, profound hearing loss & bilateral with sudden onset
  10. too much bilirubin in the blood
    -hearing loss occurs in sever cases with kernicterus
  11. gene exists on one off 22 pairs of non-sex determining chromosomes
    -hearing loss bc of heritence for EITHER parent
    -50% chance of any child inheriting hearing loss
    ex
    punett sq of Dd with dd
  12. tinnitus & dizziness common symptoms
  13. 20% of all genetic hearing loss
  14. -excess fluid in the inner ear
    -progressive or fluctuating, low-frequency HL
    -bilateral or unilateral (thought to be more often unilateral)
  15. episodic vertigo **
    tinnitus, feeling of fullness in the ear
  16. 1. usher's syndrome
    2. pendred's syndrome
    3. enlarged vestibular aqueduct syndrome
  17. attack inner ear through bloodstream---high frequencies affected first
    -aminoglycoside antibiotics (streptomycin[vestubulo toxin], neomycin, gentamicin, kanamycin, tobramycin [cochlea toxin)
    -diuretics (especially furosemide)
    -chemotherapeutic agents (platinum based agents, e.g., cisplatinin, carboplatin)
  18. 30%
  19. passed from the mother to the child via the bloodstream and/or contact with the birth canal; most common viral disease known to cause hearing loss
    -90% of symptomatic children who survive will exhibit complications, the majority will have hearing loss
    -HL is usually progressive, less often unilateral than bilateral, most often severe to profound
  20. -affects cochlea, but can also affect auditory nerve and CANS
    -high frequencies affected first
    -bilateral, symmetrical, typically mild to moderate hearing loss
  21. caused by parasite transmitted to child via placenta (not viral, it's a paraiste)
  22. benign, slow-growing tumor compressing the nerve [compresses brain stem functions]
    -(must be removed surgically)
    -most common tumor of temporal bone
  23. 2-3% of genetic hearing loss
    -rare
  24. postnatal infections; common causes of unilateral sever-profund sensorineural
  25. symptom of Pendred syndrome, associated with mondini cochlear dysplasia
    -'reflux' of endolymph through abnormally large endolymphatic duct and vestibular aqueduct (bony canal leading from inner ear to cranial cavity) causes damage to cochlear structures in scala media (akin to meniere's mechanism), endolymphatic sac thought to regulate fluid pressure—increase in CSF pressure may cause reflux ('blowing a trumpet', weight lifting, diving, air plane flight) (someitme progressive, sometimes suddenly)
    -initially in high frequencies
  26. gene present in both parents
    -25% change of any child inheriting hearing loss
    -children can be carriers
  27. children with sneorineural hearing loss of unknonw etiology
    -often bilatreal
    -sensorineural hearing loss at birth or onset in childhood
    -
  28. 1/2
  29. age-related hearing loss (50+)
  30. Toxoplasmosis
    Rubella
    Cytomegalovirus
    Herpes
    Ssyphilis
  31. genetic (endogenous)
  32. 80$ of all genetic hearing loss
    -the majority
  33. sexually transmitted, passed to fetus in utero
  34. genetic causes and otitits media
  35. -Congenital Infections: TRCHs
    -hyperbilirubinemia with kernicterus
    -trauma at birth
    -medication during birht
    -low birth weight bc of byperbilirubinemia
    -postnatal infections: bacterial mningitis, mumps, measles (rubeola)
    -ototoxic medications
    -meniere's disease
    -noise exposure
    -prescbycusis
    -genetic causes
    -otitis media
  36. -autosomar dominant inheritance
    -autosomal recessive inheritcane
    -x-linked inhertitance (passed from mother to son)
  37. 3:2
  38. maternal-contracted, passed to child through placenta prenatally; typically bilateral, severe to profound loss, with worst hearing in the mid frequencies (cookie bite configuration)

    -earlier infection increases the chance of hearing loss
    -possibly accompanied by heart/kidney defects, visual impairment (german measles)
  39. autosomal dominant inheritance; absence of organ of corti and atrophy of spiral ganglion; accompanied by:
    -white forelock of hair
    -iris bicolor or heterochromia
  40. vestibular impaiment
  41. -almost always unilateral
    -affects high frequencies first
  42. -Almost always bilateral (unless very mild), but can be asymmetrical
    -Affects frequencies between 3000 & 6000 Hz first (doesn't affect outer/middle ear, it affects cochlear
    -Notched configuration
  43. congenital deafness and progressive loss of vision (retinitis pigmentosa) leading to eventual blindness
  44. noise exposure and presbycusis
  45. 5-20%
  46. postnatal infections; rubeola
  47. -ototoxic medications
    -meniere's desease
    -noise exposure
    -presbycusis
    -genetic causes
    -otitis media
  48. postnatal infection; leading cause of acquired childhood severe sensorineural hearing loss
    -can affect cochlear, audiotry nerve, CANS (inflammation causes ossification inside cochlear)